Emily L Deer
About
Emily Deer is a researcher based in Lafayette, Louisiana, whose work focuses on the immunological and physiological mechanisms underlying hypertensive disorders of pregnancy, including preeclampsia and gestational diabetes. Her published investigations examine the roles of immune cells, particularly CD4+ T cells and CD19+ B cells, in driving elevated blood pressure during pregnancy, as well as the downstream effects of placental ischemia, oxidative stress, and mitochondrial dysfunction on maternal and fetal outcomes.
Across 38 peer-reviewed publications, Deer has contributed to journals including Hypertension in Pregnancy, the Journal of Reproductive Immunology, the American Journal of Physiology, and the American Journal of Hypertension. Her research addresses both mechanistic questions and potential therapeutic targets, with studies exploring interventions such as metformin, mitotempo, and interleukin-1 receptor inhibition in animal models of preeclampsia. Additional work has investigated developmental programming of hypertension in offspring and the relationship between hypertensive pregnancy and cognitive dysfunction.
Her body of work spans polycystic ovary syndrome, immune dysregulation in pregnancy, and the broader intersection of cardiovascular and reproductive physiology, reflecting a sustained focus on conditions that carry significant maternal and perinatal risk.
Credentials
Publications
- Placental CD4⁺ T cells from women with gestational diabetes recapitulate disease features in a pregnant rat model, improved by metformin or mitotempo.
- Immune Cells in Preeclampsia.
- AT1-AA induced hypertension during pregnancy contributes to developmental programming of hypertension in female offspring.
- Interleukin-1 receptor type 1 inhibition improves blood pressure, fetal growth and immune balance in placental ischemic rats.
- The Role of Mitochondrial Dysfunction and Oxidative Stress in Women's Reproductive Disorders: Implications for Polycystic Ovary Syndrome and Preeclampsia.
- Hypertensive Pregnancy Disorders: From Mechanisms to Management.
- Preeclamptic Placental CD19+ B Cells Are Causal to Hypertension During Pregnancy.
- Hypertension and Cognitive Dysfunction in a Pregnant Rat Model of PE; a Role for CD4+ T Cells.
- The role of T cell stimulated agonistic autoantibodies to the angiotensin II type I receptor (AT1-AA) in mediating multiorgan dysfunction in IL-17 induced hypertension during pregnancy.
- AT1-AA Is Produced in Offspring in Response to Placental Ischemia and Is Lowered by B-Cell Depletion Without Compromising Overall Offspring Health.
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